A history of excessive carotene intake consistent with the diagnosis of hypercarotenemia is usually present. Metabolic carotenemia without a history of excessive carotene intake may be due to a genetic defect in the metabolism of carotenoids. As previously mentioned, amenorrhea may be associated with carotenemia. This occurs in patients who consume a pure or predominantly vegetarian diet without red meat.
|Published (Last):||25 October 2015|
|PDF File Size:||9.24 Mb|
|ePub File Size:||4.43 Mb|
|Price:||Free* [*Free Regsitration Required]|
Yasser Al Nasser; Mohammed Albugeaey1. Affiliations Last Update: December 6, Beta-Carotene is the main carotenoid found in plants and is most responsible for this condition. Although benign, this condition is often confused with jaundice, leading to unnecessary investigations. Etiology Ingestion is the main source of carotenoids in the human body.
Fruits include apricot, cantaloupe, mango, orange, papaya, peaches, and prunes. Other food items with high carotene include butter, eggs, milk, and palm oil. The authors commented it might be due to the different mode and type of feeding they received compared to healthy children. Beta-carotene is the main carotenoid found in plants. The following are the main items that should be addressed in the patient interview and physical examination.
Evaluation A good history and physical exam help narrow down the laboratory investigation necessary. Extensive investigations are seldom needed. Often, the yellow pigmentation resolves with regulating blood sugar. Examples include saffron, quinacrine, tetryl, picric acid, and dinitrophenol.
Enhancing Healthcare Team Outcomes Patient education in collaboration with a dietitian improves the outcome of the condition and prevent further unnecessary investigations.
Questions To access free multiple choice questions on this topic, click here. References 1. Leung AK. Benign carotenemia in children. Can Fam Physician. Carotenemia in infancy and its association with prevalent feeding practices. Pediatr Dermatol. Hypercarotenaemia or hypercarotenoidaemia. Rock CL. Carotenoids: biology and treatment. Lascari AD. A review. Clin Pediatr Phila. Carotenaemia with low vitamin A levels and low retinol-binding protein. Priyadarshani AMB. Insights of hypercarotenaemia: A brief review.
Carotenaemia in children is common and benign: most can stay at home. Scott Med J. Carotenemia in mentally retarded children. Incidence and etiology. Can Med Assoc J. Sharman IM. Sale TA, Stratman E. Carotenemia associated with green bean ingestion. Vitamin A Absorption, Storage and Mobilization.
Yellow palms and soles: A rare skin manifestation in diabetes mellitus. Indian J Endocrinol Metab. Serum beta-carotene in anorexia nervosa patients: a case-control study. Int J Eat Disord. Hypercarotenaemia in anorexia nervosa]. J Dtsch Dermatol Ges. Crisp AH, Stonehill E. Hypercarotenaemia as a symptom of weight phobia. Postgrad Med J. Lycopenemia: a variant of carotenemia. Hypercarotenaemia in a tomato soup faddist.
Systemic AL amyloidosis with unusual cutaneous presentation unmasked by carotenoderma. Hyper-beta-carotenemia unrelated to diet: a case of brain tumor. Int J Vitam Nutr Res. Nishimura T. A correlation between carotenemia and biliary dyskinesia. Canthaxanthin retinopathy: long-term observations.
Ophthalmic Res. This book is distributed under the terms of the Creative Commons Attribution 4.
Pancreas disease Increased need for vitamin A should be determined by your health care professional. Claims that beta-carotene is effective as a sunscreen have not been proven. Although beta-carotene supplements are being studied for their ability to reduce the risk of certain types of cancer and possibly heart disease, there is not enough information to show that this is effective. Beta-carotene may be used to treat other conditions as determined by your doctor. Beta-carotene is available without a prescription. Importance of Diet For good health, it is important that you eat a balanced and varied diet.
Menu Carotenemia Carotenemia is a clinical condition with yellow pigmentation of the skin associated with increased blood carotene levels. In most instances it is associated with large consumption of carotene in the diet. It has been confused with jaundice. Carotene is derived from plants and it is the chief precursor of Vitamin A. Carotene is converted to Vitamin A in the mucosal cells of the small intestine. Absorption is aided by pancreatic lipase, bile salts, fat, and thyroid hormone.
Carotenemia (Yellow Skin from High Carotene Levels)
Carotenoids are a normal part of our diet and contribute to the normal colour of our skin. They also help protect our skin from sunburn. Carotenoids are precursors of an essential vitamin, Vitamin A or retinol. Conversion takes place in the mucosal cells that line the small intestine and in the liver. Pancreatic lipase enzymes, bile salts, fat, and thyroid hormone aid conversion of carotenoids to vitamin A.
Yasser Al Nasser; Mohammed Albugeaey1. Affiliations Last Update: December 6, Beta-Carotene is the main carotenoid found in plants and is most responsible for this condition. Although benign, this condition is often confused with jaundice, leading to unnecessary investigations. Etiology Ingestion is the main source of carotenoids in the human body. Fruits include apricot, cantaloupe, mango, orange, papaya, peaches, and prunes. Other food items with high carotene include butter, eggs, milk, and palm oil.